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COMPOUND RECORD / SEMAX

SemaxСемакс

CognitionPerformance
Source linked
Research profile — evidence review incomplete. Topic tags are research navigation, not indications or recommendations. Source access does not establish effectiveness or safety.
01 / OVERVIEW

What is it?

A synthetic peptide based on a fragment of adrenocorticotropic hormone (ACTH). Its research focuses on brain function and responses to neurological injury.

Identity / background source ↗

Research context

A 1997 comparative clinical report investigated Semax alongside conventional treatment in acute ischemic stroke. This is not a study of healthy-person cognitive enhancement.

Working classification
Peptide
Research collection
Neuropeptide research · editorial grouping
Institutional provenance
Not established for this compound record
Last evidence review
Full evidence review pending; source-check scope appears with each publication below.
02 / EVIDENCE DIMENSIONS

What do we know?

Human research
—Clinical comparison
Study methods
—Abstract-level review
Independent replication
—Not assessed
Source access
—English abstract
Safety evidence
—Not assessed

These are independent review fields, not a rating. Missing evidence in this starter does not mean no research exists.

03 / SOURCE TRAIL

Linked sources

Explore the research map and unresolved leads ↗

2013ReviewEnglish abstract

Neuroprotective effects of peptides bioregulators in people of various age

R. S. Umnov, N. S. Linkova, V. Kh. Khavinson · Adv Gerontol. 26(4):671–678. PMID 24738258

A gateway review comparing polypeptide complexes (Cortexin and Cerebrolysin) with short peptides (Semax, Cortagen and Pinealon). It discusses older populations and cellular mechanisms; the abstract supplies no trial-quality assessment.

Population / setting
Review of earlier research
What was checked
Indexed English abstract checked; Russian full text not retrieved.
Limitations & review scope
  • Source-linked does not mean a completed risk-of-bias review.
  • A review is not an independent clinical replication; proposed mechanisms do not establish efficacy or safety.

Source checked 2026-09-27. This is a source-linked record, not a completed evidence review.

Read the source ↗
1997Clinical trial / comparisonEnglish abstract

Effectiveness of semax in acute period of hemispheric ischemic stroke (a clinical and electrophysiological study)

E. I. Gusev, V. I. Skvortsova, N. F. Miasoedov, V. N. Nezavibatko, E. Iu. Zhuravleva, A. V. Vanichkin · Zh Nevrol Psikhiatr Im S S Korsakova. 97(6):26–34

The report studied Semax alongside intensive stroke care and described neurological and electrophysiological changes.

Population / setting
30 Semax-treated patients; 80 conventional-treatment controls
What was checked
PubMed record and English abstract checked.
Limitations & review scope
  • The abstract does not establish random allocation or blinding.
  • Disease-specific adjunctive care does not demonstrate healthy-person cognitive enhancement.
  • Full results, adverse-event reporting, and independent replication require review.

Source checked 2026-09-27. This is a source-linked record, not a completed evidence review.

Read the source ↗
Browse bibliography & PDFs ↗