Tumor-modifying effect of cardiogen peptide on M-1 sarcoma in senescent rats
Levdik NV, Knyazkin IV · 2009 · Bulletin of experimental biology and medicine 2009
Reported findings and study design
Cardiogen inhibited tumor growth in a dose-dependent pattern, with more tumor-cell apoptosis and hemorrhagic necrosis. The authors linked this to tumor vasculature rather than a direct cytostatic effect.
Senescent rats with transplanted M-1 sarcoma.
| Measure | Result | Comparison, timing and statistics |
|---|---|---|
| Tumor response | Dose-dependent growth inhibition and increased apoptosis reported; numerical magnitude not supplied |
An experimental rat tumor study; the abstract supplies no tumor-volume difference, dose schedule or human cancer outcomes.
Source for the extracted data ↗ · Checked 2026-11-01
Molekul summary of the linked publication. Educational information, not medical advice. Seek qualified healthcare help for medical conditions.
Methods, limitations & source review
Study limitations
- An experimental rat tumor study; the abstract supplies no tumor-volume difference, dose schedule or human cancer outcomes.
Publication counts are not counts of independent trials. Reviews, translations and reports sharing participants may overlap. Findings apply to the studied preparation, population and endpoints.
Review depth: Indexed abstract checked
Source access and review scope
Original-publication metadata and indexed abstract checked in Europe PMC. No unavailable full-text figures inferred.
Recorded source-check date: 2026-11-01. This date does not imply a completed systematic review.
Source type: Journal publication
Citation and original source
Original source: Levdik NV, Knyazkin IV. Tumor-modifying effect of cardiogen peptide on M-1 sarcoma in senescent rats Bulletin of experimental biology and medicine 2009 2009.
Molekul research summary: https://molekul.io/research/thin-20396706. Cite the original publication for its findings and this page when referring to Molekul’s summary or evidence appraisal.
Bibliography entry